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The effects of vagotomy on the pharmacological response to noradrenaline (NA) have been studied in the frog palate in vitro. Vagal stimulation resulted in a lower threshold of contractions to NA, and a higher maximum effect induced by this amine. The beta-adrenoceptor antagonist propranolol (PROL) antagonized the effects of vagotomy on the threshold of contractions to NA, but the antagonist was less efficient in inhibition of the maximum response. PROL antagonized the effect of vagotomy on the maximum response only when the tissue was pretreated with reserpine. A slight antagonism was obtained when the frog palate was pretreated with propranolol before noradrenaline. Pretreatment with an alpha-antagonist (phentolamine) always abolished the effect of vagotomy. The alpha 1-agonist, cirazoline (CIRZ), induced a marked contraction on the frog palate which was inhibited by the alpha 1-antagonist, d-eptyline (DET) and almost abolished by the alpha 2-antagonist, yohimbine (YOH). However, YOH

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